Acute Motor Axonal Neuropathy in Association with Hepatitis E

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Acute Motor Axonal Neuropathy in Association with Hepatitis E

Guillain-Barré syndrome (GBS) is an acute peripheral neuropathy that develops as a result of post-infectious immune-mediated nerve injury. It can be classified into classic and variant GBS. Acute motor axonal neuropathy (AMAN) is a subtype of GBS with the key clinical features of pure motor weakness, areflexia, absence of sensory symptoms, and lack of neurophysiologic evidence of demyelination....

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Acute motor axonal neuropathy

By Sameera Salman Ghauri (Dr. Ghauri of University of Texas Houston Health Science Center has no relevant financial relationships to disclose.) Suur Biliciler MD (Dr. Biliciler of University of Texas Houston Health Science Center has no relevant financial relationships to disclose.) Thy Nguyen MD (Dr. Nguyen of University of Texas Health Science Center has no relevant financial relationships to...

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Conduction block in acute motor axonal neuropathy.

Guillain-Barré syndrome is divided into two major subtypes, acute inflammatory demyelinating polyneuropathy and acute motor axonal neuropathy. The characteristic electrophysiological features of acute motor axonal neuropathy are reduced amplitude or absence of distal compound muscle action potentials indicating axonal degeneration. In contrast, autopsy study results show early nodal changes in ...

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Hyperreflexia in acute motor axonal neuropathy

Acute motor axonal neuropathy (AMAN) is a type of GBS accompanied by peripheral axonal involvement where acute motor paralysis, loss of reflex or hyporeflexia, insignificant sensory loss, and albumino-cytologic disproportion in cerebrospinal f luid are seen with no demyelinating findings in electromyography (EMG).[1] Commonly known as acute inflammatory demyelinating polyneuropathy (AIDP), GBS’...

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acute motor axonal neuropathy (aman) with motor conduction blocks in childhood; case report

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ژورنال

عنوان ژورنال: Frontiers in Neurology

سال: 2018

ISSN: 1664-2295

DOI: 10.3389/fneur.2018.00062